1. AkihiroMasumotoetal.(2001).Effect of Pravastatin on Endothelial Function in Patients With Coronary Artery Disease (Cholesterol- Independent Effect of Pravastatin). The American Journal of Cardiology. 88(11), 1291–1294.
2. Auerbach, O. et al. (1968). Thickening of Walls of Arterioles and Small Arteries In Relation to Age and Smoking Habits. The New England Journal of Medicine. 278(18), 980–984.
4. Caballero, A.E. et al. (1999). Microvascular and macrovascular reactivity is reduced in subjects at risk for type 2 diabetes. Diabetes. 48(9), 1856–1862.
5. Egashira, K. et al. (1993). Effects of age on endothelium-dependent vasodilation of resistance coronary artery by acetylcholine in humans. Circulation. 88(1), 77–81.
7. Gao,Z.etal.(2012).Altered coronary vascular control during cold stress in healthy older adults.American Journal of Physiology - Heart and Circulatory Physiology. 302(1), 312–318.
8. Garrone, P. et al. (2009). Quantitative coronary angiography in the current era: Principles and applications. Journal of Interventional Cardiology. 22(6), 527–536.
9. Gaspardone A et al. (2002). The Enhanced Vasoreactivity of the Culprit an Increased Local Release of Endothelin-1. Journal of Clinical and Basic Cardiology. 5(1), 87–92.
10.
10. Hadi, H.A.R. et al. (2005). Endothelial dysfunction: cardiovascular risk factors, therapy, and outcome. Vascular health and risk management. 1(3), 183–198.
11.
11. JassimAlSuwaidietal.(2001).Obesity is independently associated with coronary endothelial dysfunction in patients with normal or mildly diseased coronary arteries. Journal of the American College of Cardiology. 37(6), 1523–1528.
12. KangL.S.,ReyesR.A.andMuller-DelpJ.M.(2009).Aging impairs flow-induced dilation in coronary arterioles: Role of NO and H2O2. American Journal of Physiology - Heart and Circulatory Physiology. 297(3), H1087–H1095.
13. Maron D.J., Hochman J.S., Reynolds H.R., et al. (2020). Initial invasive or conservative strategy for stable coronary disease. New England Journal of Medicine. 382(15), 1395–1407.
14. Ong, P. et al. (2008). Coronary Artery Spasm as a Frequent Cause of Acute Coronary Syndrome. The CASPAR (Coronary Artery Spasm in Patients With Acute Coronary Syndrome) Study. Journal of the American College of Cardiology. 52(7), 523–527.
16. Tomai,F.etal.(2001).Unstable angina and elevated C-reactive protein levels predict enhanced vasoreactivity of the culprit lesion. Circulation. 104(13), 1471–1476.
17. Treasure, C.B. et al. (1993). Hypertension and left ventricular hypertrophy are associated with impaired endothelium-mediated relaxation in human coronary resistance vessels. Circulation. 87(1), 86–93.